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dc.contributor.authorZoccal, Daniel Breseghello [UNESP]
dc.date.accessioned2015-08-06T16:12:57Z
dc.date.available2015-08-06T16:12:57Z
dc.date.issued2015
dc.identifierhttp://onlinelibrary.wiley.com/doi/10.1113/expphysiol.2014.079558/full
dc.identifier.citationExperimental Physiology, v. 100, n. 2, p. 143-148, 2015.
dc.identifier.issn0958-0670
dc.identifier.urihttp://hdl.handle.net/11449/125727
dc.description.abstractChronic intermittent hypoxia (CIH) has been identified as a relevant risk factor for the development of enhanced sympathetic outflow and arterial hypertension. Several studies have highlighted the importance of peripheral chemoreceptors for the cardiovascular changes elicited by CIH. However, the effects of CIH on the central mechanisms regulating sympathetic outflow are not fully elucidated. Our research group has explored the hypothesis that the enhanced sympathetic drive following CIH exposure is, at least in part, dependent on alterations in the respiratory network and its interaction with the sympathetic nervous system. In this report, I discuss the changes in the discharge profile of baseline sympathetic activity in rats exposed to CIH, their association with the generation of active expiration and the interactions between expiratory and sympathetic neurones after CIH conditioning. Together, these findings are consistent with the theory that mechanisms of central respiratory–sympathetic coupling are a novel factor in the development of neurogenic hypertension.en
dc.format.extent143-148
dc.language.isoeng
dc.relation.ispartofExperimental Physiology
dc.sourceCurrículo Lattes
dc.titlePeripheral chemoreceptors and cardiorespiratory coupling: a link to sympatho-excitationen
dc.typeArtigo
dc.contributor.institutionUniversidade Estadual Paulista (UNESP)
dc.description.affiliationUniversidade Estadual Paulista Júlio de Mesquita Filho, Departamento de Fisiologia e Patologia, Faculdade de Odontologia de Araraquara, Araraquara, Rua Humaitá, 1680, Centro, CEP 14801903, SP, Brasil
dc.description.affiliationUnespUniversidade Estadual Paulista Júlio de Mesquita Filho, Departamento de Fisiologia e Patologia, Faculdade de Odontologia de Araraquara, Araraquara, Rua Humaitá, 1680, Centro, CEP 14801903, SP, Brasil
dc.identifier.doi10.1113/expphysiol.2014.079558
dc.rights.accessRightsAcesso restrito
unesp.campusUniversidade Estadual Paulista (UNESP), Faculdade de Odontologia, Araraquarapt
dc.identifier.lattes1958567557189244
unesp.departmentFisiologia e Patologiapt
unesp.author.lattes1958567557189244
dc.relation.ispartofjcr2.732
dc.relation.ispartofsjr1,238
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