Lira, F. S. [UNESP]Antunes, B. M. [UNESP]Figueiredo, C. [UNESP]Campos, E. Z. [UNESP]Panissa, V. L.G.St-Pierre, D. H.Lavoie, J. M.Magri-Tomaz, L.2019-10-062019-10-062019-03-01Diabetes and Metabolic Syndrome: Clinical Research and Reviews, v. 13, n. 2, p. 1359-1364, 2019.1878-03341871-4021http://hdl.handle.net/11449/187378Purpose: To compare the acute and chronic effects of high-intensity intermittent training (HIIT) and moderate-intensity continuous training (MICT) on indices of cardio-metabolic health: (HDL-c, total cholesterol, triglycerides, heart ratio, and phase angle/PhA) in physically active men. Methods: Twenty active men were randomly allocated to HIIT (n = 10), or MICT (n = 10) for 5 weeks, three times per week. HIIT consisted of running 5 km with 1-min at 100% of maximal aerobic speed interspersed by 1-min passive recovery while subjects in MICT group ran continuously the same 5 km at 70% of maximal aerobic speed. Blood samples were collected at different moments during the first and last exercise session. Before and after 5 weeks of both exercise training protocols, heart ratio (during exercise session) and PhA were measured pre and post-exercise training. Results: Fasting HDL-c levels did not change after 5 weeks of HIIT or MICT. Perceptual variation of HDL pre and post training (fed state) tended to differ between HIIT and MICT (p = 0.09). All lipoproteins parameters (HDL-c, total cholesterol, triglycerides and non-HDL) were increased in post-acute exercise session compared to pre-exercise during the first and last training session, these being observed after both training protocols. PhA and heart rate measured at different times during the first and last training session were not affected in both training protocols. Conclusion: These results indicate that HIIT and MICT modify the post-exercise lipoprotein profile acutely. On the other hand, only HIIT tended to increase HDL-c levels chronically.1359-1364engAcute exerciseCardiometabolic indicesCholesterolHigh-intensity trainingLipoproteinsArtigo10.1016/j.dsx.2019.02.006Acesso aberto2-s2.0-85061838742