Caffeine improved time to exhaustion but did not change alternative maximal accumulated oxygen deficit estimated during a single supramaximal running bout

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dc.contributor.authorDe Araujo Bonetti De Poli, Rodrigo [UNESP]
dc.contributor.authorMiyagi, Willian Eiji [UNESP]
dc.contributor.authorNakamura, Fabio Yuzo
dc.contributor.authorZagatto, Alessandro Moura [UNESP]
dc.contributor.institutionUniversidade Estadual Paulista (Unesp)
dc.contributor.institutionNAR-Nucleus of High Performance in Sport
dc.date.accessioned2018-12-11T16:45:42Z
dc.date.available2018-12-11T16:45:42Z
dc.date.issued2016-12-01
dc.description.abstractThe aim of the current study was to investigate the effects of acute caffeine supplementation on anaerobic capacity determined by the alternative maximal accumulated oxygen deficit (MAODALT) in running effort. Eighteen recreational male runners [29 ± 7years; total body mass 72.1 ± 5.8 kg; height 176.0 ± 5.4cm; maximal oxygen uptake (VO2max) 55.8 ± 4.2 ml·kg-1 ·min-1] underwent a graded exercise test. Caffeine (6 mg·kg-1) or a placebo were administered 1 hr before the supramaximal effort at 115% of the intensity associated with VO2max in a double-blind, randomized cross-over study, for MAODALT assessment. The time to exhaustion under caffeine condition (130.2 ± 24.5s) was 11.3% higher (p = .01) than placebo condition (118.8 ± 24.9 s) and the qualitative inference for substantial changes showed a very likely positive effect (93%). The net participation of the oxidative phosphorylation pathway was significantly higher in the caffeine condition (p = .02) and showed a likely positive effect (90%) of 15.3% with caffeine supplementation. The time constant of abrupt decay of excess postexercise oxygen consumption (τ1) was significantly different between caffeine and placebo conditions (p = .03) and showed a likely negative effect (90%), decreasing -8.0% with caffeine supplementation. The oxygen equivalents estimated from the glycolytic and phosphagen metabolic pathways showed a possibly positive effect (68%) and possibly negative effect (78%) in the qualitative inference with caffeine ingestion, respectively. However, the MAODALT did not differ under the caffeine or placebo conditions (p = .68). Therefore, we can conclude that acute caffeine ingestion does not modify the MAODALT, reinforcing the robustness of this method. However, caffeine ingestion can alter the glycolytic and phosphagen metabolic pathway contributions to MAODALT.en
dc.description.affiliationLaboratory of Physiology and Sport Performance (LAFIDE) São Paulo State University (Unesp) School of Sciences
dc.description.affiliationNAR-Nucleus of High Performance in Sport
dc.description.affiliationUnespLaboratory of Physiology and Sport Performance (LAFIDE) São Paulo State University (Unesp) School of Sciences
dc.format.extent549-557
dc.identifierhttp://dx.doi.org/10.1123/ijsnem.2016-0038
dc.identifier.citationInternational Journal of Sport Nutrition and Exercise Metabolism, v. 26, n. 6, p. 549-557, 2016.
dc.identifier.doi10.1123/ijsnem.2016-0038
dc.identifier.issn1543-2742
dc.identifier.issn1526-484X
dc.identifier.scopus2-s2.0-85010685790
dc.identifier.urihttp://hdl.handle.net/11449/169400
dc.language.isoeng
dc.relation.ispartofInternational Journal of Sport Nutrition and Exercise Metabolism
dc.relation.ispartofsjr0,963
dc.rights.accessRightsAcesso restrito
dc.sourceScopus
dc.subjectAnaerobic capacity
dc.subjectEnergetics
dc.subjectErgogenic
dc.titleCaffeine improved time to exhaustion but did not change alternative maximal accumulated oxygen deficit estimated during a single supramaximal running bouten
dc.typeArtigo
unesp.author.lattes2545518618024469[4]
unesp.author.orcid0000-0003-1065-4158[4]

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