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dc.contributor.authorCararo-Lopes, Eduardo
dc.contributor.authorDias, Matheus H.
dc.contributor.authorda Silva, Marcelo S.
dc.contributor.authorZeidler, Julianna D.
dc.contributor.authorVessoni, Alexandre T.
dc.contributor.authorReis, Marcelo S.
dc.contributor.authorBoccardo, Enrique
dc.contributor.authorArmelin, Hugo A.
dc.contributor.institutionInstituto Butantan
dc.contributor.institutionUniversidade de São Paulo (USP)
dc.contributor.institutionUniversidade do Estado de São Paulo
dc.contributor.institutionWashington University in St. Louis
dc.contributor.institutionMayo Clinic College of Medicine
dc.contributor.institutionRutgers Cancer Institute of New Jersey
dc.date.accessioned2022-04-29T08:45:30Z
dc.date.available2022-04-29T08:45:30Z
dc.date.issued2021-04-01
dc.description.abstractThe original version of this article unfortunately contained an error in the affiliations. The affiliations were not correctly assigned to the authors. The original article has been corrected.en
dc.description.affiliationCenter of Toxins Immune-response and Cell Signaling Instituto Butantan
dc.description.affiliationDepartment of Biochemistry Instituto de Química Universidade de São Paulo
dc.description.affiliationDepartment of Chemical and Biological Sciences Instituto de Biociência Universidade do Estado de São Paulo
dc.description.affiliationDepartment of Medicine Washington University in St. Louis
dc.description.affiliationDepartment of Microbiology Instituto de Biociências Universidade de São Paulo
dc.description.affiliationKogod Aging Center Department of Anesthesiology and Perioperative Medicine Mayo Clinic College of Medicine
dc.description.affiliationRutgers Cancer Institute of New Jersey
dc.identifierhttp://dx.doi.org/10.1038/s41419-021-03564-4
dc.identifier.citationCell Death and Disease, v. 12, n. 4, 2021.
dc.identifier.doi10.1038/s41419-021-03564-4
dc.identifier.issn2041-4889
dc.identifier.scopus2-s2.0-85102704314
dc.identifier.urihttp://hdl.handle.net/11449/231454
dc.language.isoeng
dc.relation.ispartofCell Death and Disease
dc.sourceScopus
dc.title.alternativeCorrection: Autophagy buffers Ras-induced genotoxic stress enabling malignant transformation in keratinocytes primed by human papillomavirus (Cell Death & Disease, (2021), 12, 2, (194), 10.1038/s41419-021-03476-3)en
dc.typeErrata
unesp.author.orcid0000-0002-5316-5146[1]
unesp.author.orcid0000-0003-4203-9299[3]
unesp.author.orcid0000-0001-9170-5185[5]
unesp.author.orcid0000-0002-3754-9115[6]
unesp.author.orcid0000-0001-9557-0804[8]

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