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The control of sodium chloride intake: Functional relationship between hypothalamic inhibitory areas and amygdaloid complex stimulating areas

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dc.contributor.authorSaad, Wilson A. [UNESP]
dc.contributor.authorCamargo, Luiz A.A. [UNESP]
dc.contributor.authorSilva-Netto, Cincinato R.
dc.contributor.authorAntunes-Rodrigues, J.
dc.contributor.authorCovian, Miguel R.
dc.contributor.institutionUniversidade Estadual Paulista (Unesp)
dc.contributor.institutionSchool of Medicine
dc.date.accessioned2014-05-26T18:26:47Z
dc.date.available2014-05-26T18:26:47Z
dc.date.issued1981-03-01
dc.description.abstractSodium chloride intake was studied in rats submitted to different neurosurgical procedures. Intake decreased in animals submitted to bilateral destruction of the basolateral amygdaloid complex, and increased after the same animals were submitted to destruction of the anterior lateral hypothalamus, a procedure which is known to cause increased intake in intact rats. In the reverse experiment, where the anterior lateral hypothalamus was destroyed before the basolateral amygdaloid complex, the effect of increased sodium chloride intake induced by destruction of the hypothalamus overcame the decreased expected upon destruction of the amygdaloid complex. These results permit us to conclude that the hypothalamic areas which inhibit sodium chloride intake predominate over the stimulating areas of the amygdaloid complex in the control of sodium chloride intake. © 1981 ANKHO International Inc.en
dc.description.affiliationDepartment of Physiology School of Dentistry UNESP, 14800 Araraquara, SP
dc.description.affiliationDepartment of Physiology School of Medicine, 14100 Ribeiräo Preto, SP
dc.description.affiliationUnespDepartment of Physiology School of Dentistry UNESP, 14800 Araraquara, SP
dc.format.extent197-202
dc.identifierhttp://dx.doi.org/10.1016/S0361-9230(81)80047-0
dc.identifier.citationBrain Research Bulletin, v. 6, n. 3, p. 197-202, 1981.
dc.identifier.doi10.1016/S0361-9230(81)80047-0
dc.identifier.issn0361-9230
dc.identifier.scopus2-s2.0-0019831471
dc.identifier.urihttp://hdl.handle.net/11449/132293
dc.identifier.wosWOS:A1981LK02300001
dc.language.isoeng
dc.publisherElsevier B.V.
dc.relation.ispartofBrain Research Bulletin
dc.relation.ispartofjcr3.440
dc.relation.ispartofsjr1,398
dc.rights.accessRightsAcesso restritopt
dc.sourceScopus
dc.subjectAmygdala
dc.subjectBehavior
dc.subjectHypothalamus
dc.subjectSelf-selection
dc.subjectSodium chloride intake
dc.subjectSodium
dc.subjectAmygdaloid nucleus
dc.subjectAnimal experiment
dc.subjectBehavior
dc.subjectBrain injury
dc.subjectCentral nervous system
dc.subjectDrug absorption
dc.subjectFeeding behavior
dc.subjectHypothalamus
dc.subjectRat
dc.subjectAnimals
dc.subjectNeural Inhibition
dc.subjectNeural Pathways
dc.subjectParaventricular Hypothalamic Nucleus
dc.subjectRats
dc.subjectSeptum Pellucidum
dc.subjectSodium Chloride
dc.subjectSupraoptic Nucleus
dc.subjectWater-Electrolyte Balance
dc.titleThe control of sodium chloride intake: Functional relationship between hypothalamic inhibitory areas and amygdaloid complex stimulating areasen
dc.typeArtigopt
dcterms.licensehttp://www.elsevier.com/about/open-access/open-access-policies/article-posting-policy
dcterms.rightsHolderElsevier B.V.
dspace.entity.typePublication
relation.isDepartmentOfPublicationb3ba3d9c-022e-4521-8805-0bcceea7372e
relation.isDepartmentOfPublication.latestForDiscoveryb3ba3d9c-022e-4521-8805-0bcceea7372e
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unesp.campusUniversidade Estadual Paulista (UNESP), Faculdade de Odontologia, Araraquarapt
unesp.departmentFisiologia e Patologia - FOARpt

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Araraquara - FOAR - Faculdade de Odontologia